Post-anoxic myoclonus

  • Pavis Laengvejkal Department of Neurology, Texas Tech University Health Sciences Center
  • Parunyou Julayanont Department of Neurology, Texas Tech University Health Sciences Center
  • Drew Payne Department of Internal Medicine, Texas Tech University Health Sciences Center
Keywords: Post-anoxic myoclonus, PAM, Lance-Adams syndrome

Abstract

Myoclonus is a movement disorder characterized by involuntary, sudden, brief muscle jerks caused by muscular contraction (positive myoclonus) or inhibition (negative myoclonus).1,2 Myoclonus is generally a medical sign and not a diagnosis. It can occur in multiple disorders. A short differential diagnosis list includes anoxic brain injury, multiple sclerosis, Parkinson's disease, subacute sclerosing panencephalitis, and Creutzfeldt-Jakob disease.  One way to classify the etiologies is through review of the clinical presentation and comorbid conditions. This article presents a review of anoxic brain injury related myoclonus. Post-anoxic myoclonus (PAM) can develop in either acute or chronic phase. Acute PAM occurs within hours after hypoxic event; chronic PAM (Lance-Adams syndrome) develops in survivors several days to weeks after the episodes of brain hypoxia.

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Published
2014-04-06
How to Cite
Laengvejkal, P., Julayanont, P., & Payne, D. (2014). Post-anoxic myoclonus. The Southwest Respiratory and Critical Care Chronicles, 2(6), 8-14. Retrieved from https://pulmonarychronicles.com/index.php/pulmonarychronicles/article/view/130